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Research paper Sera from neuromyelitis optica patients disrupt the blood–brain barrier Fumitaka Shimizu1, Yasuteru Sano1, Toshiyuki Takahashi2, Hiroyo Haruki1, Kazuyuki Saito1, Michiaki Koga1, Takashi Kanda1 + Author Affiliations 1Department of Neurology and Clinical Neuroscience, Yamaguchi University Graduate School of Medicine, Ube, Japan 2Department of Neurology, Tohoku University Graduate School of Medicine, Miyagi, Japan Correspondence to Dr T Kanda, Department of Neurology and Clinical Neuroscience, Yamaguchi University Graduate School of Medicine, 1-1-1, Minamikogushi, Ube, Yamaguchi 7558505, Japan; tkanda@yamaguchi-u.ac.jp Contributors FS and TK conceived and designed the study. All authors reviewed, amended and agreed on the final version of the manuscript.


Atypical presentations of neuromyelitis optica Douglas SatoI, II; Kazuo FujiharaIII IDepartment of Neurology, Tohoku University Graduate School of Medicine, Sendai, Japan IIDepartment of Neurology, Faculty of Medicine – University of São Paulo, São Paulo SP, Brazil IIIDepartment of Multiple Sclerosis Therapeutics, Tohoku University Graduate School of Medicine, Sendai, Japan Neuromyelitis optica (NMO) or Devic’s disease is an inflammatory disease of central nervous system classically characterized by acute, severe episodes of optic neuritis (ON) and longitudinally extensive transverse myelitis (TM)1. The article published in 1894 by Dr Eugène Devic was based on a 45-years old female patient presenting with bilateral ON followed by TM and she deceased about a month after the monophasic opticomyelitis2.


Neuromyelitis optica in a patient with pemphigus foliaceus. Salazar R, Cerghet M, Farhat E, Lim HW


Rituximab dosing and monitoring strategies in neuromyelitis optica patients: creating strategies for therapeutic success Benjamin M Greenberg Donna Graves Gina Remington Paula Hardeman Martha Mann Nitin Karandikar Olaf Stuve Nancy Monson Elliot Frohman University of Texas Southwestern Medical Center, Dallas, TX, USA Benjamin M. Greenberg, MD, MHS, University of Texas Southwestern Medical Center, 5323 Harry Hines Boulevard, Dallas, TX 75390-8806, USA. Email: benjamin.greenberg@utsouthwestern.edu Abstract Background: Neuromyelitis optica (NMO) is an autoimmune condition that predominantly causes severe optic neuritis and transverse myelitis.


Painful Tonic Spasm in Neuromyelitis Optica Incidence, Diagnostic Utility, and Clinical Characteristics Sung-Min Kim, MD; Min Jin Go, MS; Jung-Joon Sung, MD, PhD; Kyung Seok Park, MD, PhD; Kwang-Woo Lee, MD, PhD Arch Neurol.


Failure of Natalizumab to Prevent Relapses in Neuromyelitis Optica Ingo Kleiter, MD; Kerstin Hellwig, MD; Achim Berthele, MD; Tania K?mpfel, MD; Ralf A. Linker, MD; Hans-Peter Hartung, MD; Friedemann Paul, MD; Orhan Aktas, MD; for the Neuromyelitis Optica Study Group Arch Neurol. ?2012;69(2):239-245.


Interstim Therapy InterStim therapy is a pacemaker for the bladder used to reestablish appropriate nueromodulation of the bladder. The device works by using a stimulator device in the buttocks to stimulate the S3 nerve root in the pelvis. This is the primary nerve root for bladder function.


Overview Management of voiding dysfunction in neurologic disorders can substantially improve the quality of life as well as the health of patients. A systematic review of the pattern of voiding dysfunction leads to an understanding of the underlying mechanism, which, in turn, allows the treating physician to develop a strategy for managing it. Neurourology, although a relatively new field of study, has revolutionized the management of a vexing problem that affects millions of patients.


Brain Involvement in Neuromyelitis Optica Spectrum Disorders Koon Ho Chan, MD, FRCP; C. T. Tse, MBBS, MRCP; C.


Case Reports in Medicine Volume 2011 (2011), Article ID 393568, 4 pages doi:10.1155/2011/393568 Case Report Neuromyelitis Optica Spectrum Disease with Positive Autoimmune Indices: A Case Report and Review of the Literature M.


A population-based study of neuromyelitis optica in Caucasians N.


Pathogenic T cell responses against aquaporin 4. Pohl M , Fischer MT , Mader S , Schanda K , Kitic M , Sharma R , Wimmer I , Misu T , Fujihara K , Reindl M , Lassmann H , Bradl M . Source Department of Neuroimmunology, Center for Brain Research, Medical University Vienna, Spitalgasse 4, 1090, Vienna, Austria.


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